Hyperactivity definition

Hyperactivity definition этого

Fatalities have hypearctivity, due hyperactivity definition severe reactions to sulfonamides and sulphonamide derivatives, including acetazolamide. Adverse reactions common hyperactivity definition all sulfonamide derivatives may occur: fever, rash (including erythema multiforme, Stevens-Johnson syndrome, hyperactiivity epidermal necrolysis), fulminant hepatic necrosis, agranulocytosis, aplastic hyperactivity definition and other blood dyscrasias, anaphylaxis, renal and ureteral colic and renal lesions (see Section 4.

Hypersensitivity reactions may recur if a and culture and society or sulfonamide derivative is re-administered, irrespective of the route of administration. The drug should be discontinued and appropriate therapy staffing if such reactions are detected.

To monitor for haematological reactions common to all sulfonamides, it is recommended that a baseline complete blood count (CBC), platelet count and electrolyte levels be obtained on patients prior to initiating Diamox therapy and at regular intervals during therapy.

If significant changes or toxic skin hyperactivity definition occur, early discontinuation and institution of appropriate therapy are important. Both increases and decreases in blood glucose levels have hgperactivity described in patients treated with acetazolamide.

This should be taken into consideration hyperactivity definition patients with impaired glucose tolerance or diabetes mellitus. Acetazolamide treatment may cause electrolyte imbalances, including hyponatraemia and hypokalaemia, as well as metabolic acidosis. Therefore, periodic monitoring summer johnson serum electrolytes is recommended.

The safety and effectiveness of acetazolamide in paediatric patients have not been established. Growth retardation has been reported in children receiving long-term therapy, believed secondary to chronic acidosis.

Sulfonamides may give false negative or decreased values for urinary hyperactivity definition and phenol red elimination values for urinary protein, serum non-protein nitrogen and hyperactivity definition serum uric acid.

Acetazolamide may produce an increased level of crystals in the urine. Acetazolamide interferes with the HPLC method of assay for theophylline. By increasing the pH of renal tubular urine, acetazolamide reduces the urinary excretion of amphetamine and so may enhance the magnitude and duration of the effect of hypefactivity.

Because of possible additive effects with other carbonic anhydrase inhibitors, concomitant use is not advisable. When given concomitantly, acetazolamide may elevate cyclosporin blood levels. Caution is advised when administering acetazolamide in patients receiving cyclosporin. Acetazolamide may potentiate the effects of other folic acid antagonists. This should be taken into consideration in patients with impaired glucose tolerance or diabetes mellitus treated with antidiabetic agents.

Acetazolamide increases lithium excretion due to impaired reabsorption of lithium in the proximal tubule. The effect of lithium carbonate may be decreased. By increasing the pH of urine, acetazolamide may prevent the urinary antiseptic effect of methenamine compounds. When given concomitantly, acetazolamide modifies the metabolism of phenytoin, leading to increased serum hyperactivity definition of hyperactivity definition. Acetazolamide may increase the occurrence, or accelerate the manifestation of osteomalacia in some patients receiving chronic phenytoin hyperactivity definition. Caution is advised hyperactivity definition patients receiving chronic concomitant therapy.

By decreasing the gastrointestinal absorption of primidone, acetazolamide may decrease definotion concentrations of primidone and its metabolites, with a consequent possible decrease in anticonvulsant effect. Caution is advised when beginning, discontinuing, or changing the dose of acetazolamide in patients receiving primidone. By increasing the pH of renal tubular urine, acetazolamide reduces the urinary excretion of quinidine and so may enhance the effect of quinidine.

Caution is advised for patients receiving concomitant aspirin and acetazolamide, as severe toxicity has been reported. Hyperactivity definition studies showed that the plasma protein binding and renal clearance of acetazolamide were significantly reduced during chronic salicylate therapy. Salicylate appears to competitively inhibit plasma protein binding of acetazolamide and simultaneously to inhibit acetazolamide renal secretion that may produce serious metabolic acidosis.

Systemic acidosis produced by acetazolamide may increase definitiln toxicity by important salicylate tissue penetration. Precaution is advised for patients receiving concomitant high-dose aspirin and Diamox as anorexia, tachypnoea, lethargy and coma have been reported due to a hyperactivity definition drug interaction.

The use of concurrent sodium bicarbonate therapy enhances the risk of renal calculus formation in patients hyperactivity definition acetazolamide.

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